2009年9月9-12日
中国·杭州
   
 

第一届世界天然药物和传统药物药理学学术会议第二轮通知
征集论文

1. 征文截止日期:2009年6月20日,会议论文在中国药理学会核心期刊《中国药理学与毒理学杂志》上刊登,逾期则无法刊载。此外,截至6月20日未交纳会议注册费者论文摘要将不予刊载。摘要通过e-mail提交到如下地址: zhxd@imm.ac.cn
2. 摘要撰写格式
摘要包括如下内容:
itle: Not more than 150 characters including spaces.
uthors:
> Enter all the co-authors’ names. The writing order is first name followed by the family name (family name in capital letters). The participant may be the first presenting author of one abstract only, however, a participant may be second or subsequent author of other abstracts.
ffiliations: Enter the institutions for all your coauthors.
bstract
> The body of the abstract (include key words and acknowledgement) should be no more than 1600 characters including spaces.
> Each abstract should contain a sentence stating the study objective; a brief statement of methods; a summary of the results obtained; and a statement of the conclusions. Use standard abbreviations for units of measure. Other abbreviations should be spelled out in full at the first mention, followed by the abbreviation in parentheses.
-- Key words: The key words should be no more than four and presented in one line.
─ Acknowledgement: Thanks should follow the key words at the end of the abstract.
─ Example
Neuroprotective effect of taurine against acute cortical neurons injury induced by oxygen-glucose deprivation
Can LUO1, Lian-Jun GUO1, Zhi-Kai DAI2, Qin WU2, Jing-Shan SHI2
(1. Department of Pharmacology, Tongji Medical College, Huazhong University of Science and Technology, Wuhan 430030, China; 2. Department of Pharmacology, Zunyi Medical College, Zunyi 563003, China)
Abstract: AIM Taurine was reported neuroprotective under several ischemic models in vivo. In this study, the direct effect of taurine against oxygen-glucose deprivation (OGD) inducing acute neuronal injury and the underlying mechanisms in vitro were investigated. METHODS Four hours OGD was used to induce in vitro ischemic injury in rat cortical neuron. Taurine 5, 10 and 20 mmol·L-1 was added 20 h before and during 4 h OGD period respectively. Mortality rate of neuron was assayed by MTT and flow cytometry methods. Level of neuronal intracellular free Ca2+ concentration ( [Ca2+]i ) was detected by Fura 2/AM loading. Amino acid concentrations in culture media were measured by high performance liquid chromatography. RESULTS Under OGD conditions, neuronal death was markedly increased, and the levels of neuronal [Ca2+]i and extracellular glutamate were enhanced obviously. Taurine pretreatment obviously decreased the percentage of neuronal death induced by OGD. In addition, abnormal elevation of neuronal [Ca2+]i and extracellular glutamate level induced by OGD both were markedly repressed by taurine. CONCLUSION Taurine can alleviate rat cortical neuron injury induced by OGD, the mechanisms were likely due to repressing calcium overload and inhibiting excessive release or leakage of glutamate under such conditions.
Key words: taurine; neurons;oxygen glucose deprivation; calcium, cytosolic; apoptosis
3. 摘要通过e-mail提交到如下地址: zhxd@imm.ac.cn
4. 摘要可以壁报的形式展出,壁报尺寸为110 cm(长),90 cm(宽)。


  中国药理学会
联系方式:
联系人:赵小丹 刘燕平
地址:北京市宣武区先农坛街1号,中国药理学会办公室,100050
电话:010-63165211,13671024448
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电子信箱:zhxd@imm.ac.cn