1. The deadline for the reception of abstracts will be May 31, 2009
2. Format of the abstract
The abstract include the following information:
Title: Not more than 150 characters including spaces.
Authors:
> Enter all the co-authors’ names. The writing order is first name followed by the family name (family name in capital letters). The participant may be the first presenting author of one abstract only, however, a participant may be second or subsequent author of other abstracts.
Affiliations: Enter the institutions for all your coauthors.
Abstract
> The body of the abstract (include key words and acknowledgement) should be no more than 1600 characters including spaces.
> Each abstract should contain a sentence stating the study objective; a brief statement of methods; a summary of the results obtained; and a statement of the conclusions. Use standard abbreviations for units of measure. Other abbreviations should be spelled out in full at the first mention, followed by the abbreviation in parentheses.
-- Key words: The key words should be no more than four and presented in one line.
─ Acknowledgement: Thanks should follow the key words at the end of the abstract.
─ Example
Neuroprotective effect of taurine against acute cortical neurons injury induced by oxygen-glucose deprivation
Can LUO1, Lian-Jun GUO1, Zhi-Kai DAI2, Qin WU2, Jing-Shan SHI2
(1. Department of Pharmacology, Tongji Medical College, Huazhong University of Science and Technology, Wuhan 430030, China; 2. Department of Pharmacology, Zunyi Medical College, Zunyi 563003, China)
Abstract: AIM Taurine was reported neuroprotective under several ischemic models in vivo. In this study, the direct effect of taurine against oxygen-glucose deprivation (OGD) inducing acute neuronal injury and the underlying mechanisms in vitro were investigated. METHODS Four hours OGD was used to induce in vitro ischemic injury in rat cortical neuron. Taurine 5, 10 and 20 mmol·L-1 was added 20 h before and during 4 h OGD period respectively. Mortality rate of neuron was assayed by MTT and flow cytometry methods. Level of neuronal intracellular free Ca2+ concentration ( [Ca2+]i ) was detected by Fura 2/AM loading. Amino acid concentrations in culture media were measured by high performance liquid chromatography. RESULTS Under OGD conditions, neuronal death was markedly increased, and the levels of neuronal [Ca2+]i and extracellular glutamate were enhanced obviously. Taurine pretreatment obviously decreased the percentage of neuronal death induced by OGD. In addition, abnormal elevation of neuronal [Ca2+]i and extracellular glutamate level induced by OGD both were markedly repressed by taurine. CONCLUSION Taurine can alleviate rat cortical neuron injury induced by OGD, the mechanisms were likely due to repressing calcium overload and inhibiting excessive release or leakage of glutamate under such conditions.
Key words: taurine; neurons；oxygen glucose deprivation; calcium, cytosolic; apoptosis

3. The abstract should be submitted by e-mail to the following address:
zhouwx@nic.bmi.ac.cn; jennifer-jn@126.com

4. All the abstract will be presented in poster format, except for those selected as oral presentations. The format of the poster is 110 cm high and 80 cm wide.
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